Metabolic alterations linked to T2D are well characterised by epidemiological and research based mostly research. The pathogenesis and progression of T2D is ascribed to four mechanisms; amplified advanced glycation end product (AGE) development, greater polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[7]. Till https://www.directivepublications.org/journal-of-diabetology-research/